D-solve - Solve Diabetes Using Low Carb & Low Insulin Diabetes Management

Mainstream nutritional science has demonized dietary fat, yet 50 years and hundreds of millions of dollars of research have failed to prove that eating a low-fat diet will help you live longer.

ABSTRACTThere is growing awareness that the profound changes in the environment (eg, in diet and other lifestyle conditions) that began with the introduction of agriculture and animal husbandry
10 000 y ago occurred too recently on an evolutionary time scale for the human genome to adjust. In conjunction with this discordance between our ancient, genetically determined biology and the nutritional, cultural, and activity patterns of contemporary Western populations, many of the so-called diseases of civilization have emerged. In particular, food staples and food-processing procedures introduced during the Neolithic and Industrial Periods have fundamentally altered 7 crucial nutritional characteristics of ancestral hominin diets: 1) glycemic load, 2) fatty acid composition, 3) macronutrient composition, 4) micronutrient density, 5) acid-base balance, 6) sodium-potassium ratio, and 7) fiber content. The evolutionary collision of our ancient genome with the nutritional qualities of recently introduced foods may underlie many of the chronic diseases of Western civilization. Am J Clin Nutr 2005;81:341–54.

The persistence of an epidemic of obesity and type 2 diabetes suggeststhat new nutritional strategies are needed if the epidemic is tobe overcome. A promising nutritional approach suggested by thisthematic review is carbohydrate restriction. Recent studies showthat, under conditions of carbohydrate restriction, fuel sources shiftfrom glucose and fatty acids to fatty acids and ketones, and that adlibitum–fed carbohydrate-restricted diets lead to appetite reduction,weight loss, and improvement in surrogate markers of cardiovasculardisease. Am J Clin Nutr 2007;86:276–84.

ABSTRACTThe epidemics of obesity, metabolic syndrome and type 2 diabetes have worsened over the past decades. During this time our preventive and therapeutic approach (the ‘vaccine’), consisting of a low-fat diet and exercise, has remained fundamentally unchanged. A case is made that these conditions are inter-related and may be caused by a single underlying factor related to the carbohydrate content of diet. The validity of the present approach is challenged when those most knowledgeable in its application succumb to diseases it is meant to prevent. Others argue against the status quo that a low-carbohydrate diet may be more beneficial. A strong belief in the present approach discouraged research into low-carbohydrate diets until recently. Several studies have now demonstrated their benefits and are refuting old claims that they cause harm. Aboriginal people suffer more acutely from the epidemics in question and their dietary history suggests that a sudden increase in carbohydrates is to blame. Recent studies and a case history demonstrate that carbohydrate consumption can drive appetite and over-eating while carbohydrate restriction leads to weight loss and improvement in the markers for metabolic syndrome and type 2 diabetes. The growing evidence in support of low-carbohydrate diets will encounter resistance from economic interests threatened by changes in consumption patterns.

Objective: Various studies have convincingly shown the beneficial effect of ketogenic diet (in which the daily consumption of carbohydrate is less than 20 grams, regardless of fat, protein and caloric intake) in reducing weight in obese subjects. However, its long term effect on obese subjects with high total cholesterol (as compared to obese subjects with normal cholesterol level) is lacking. It is believed that ketogenic diet may have adverse effect on the lipid profile. Therefore, in this study the effect of ketogenic diet in obese subjects with high cholesterol level above 6 mmol/L is compared to those with normocholesterolemia for a period of 56 weeks.Results: The body weight and body mass index of both groups decreased significantly (P < 0.0001). The level of total holesterol, LDL cholesterol, triglycerides and blood glucose level decreased significantly (P < 0.0001), whereas HDL holesterol increased significantly (P < 0.0001) after the treatment in both groups.Conclusion: This study shows the beneficial effects of ketogenic diet following its long term administration in obese subjects with a high level of total cholesterol. Moreover, this study demonstrates that low carbohydrate diet is safe to use for a longer period of time in obese subjects with a high total cholesterol level and those with normocholesterolemia.